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“Introduction Acute mesenteric ischemia (AMI) can result from vascular occlusive or non-occlusive conditions. Non-occlusive mesenteric ischemia is caused by conditions such as hypovolemia, sepsis, and cardiogenic shock, whereas the underlying cause of ischemia in 70–80% of cases with AMI is the occlusion of the superior mesenteric artery, caused by embolism or thrombosis [1]. Irreversible changes in the bowel mucosa occur within 6 h in the case of acute arterial occlusion, leading to the disruption of the mucosal barrier, which subsequently allows bacterial translocation, peritonitis, sepsis, and rapid progression to multiple stiripentol organ failure. Bowel ischemia and necrosis develop rapidly due to a lack of sufficient

time to develop collateral circulation, particularly in cases with embolism. Despite advances in diagnosis, treatment, and post-operative care in recent years, AMI still has a high mortality rate, ranging between 40 and 70% [2]. The most important causes of the high mortality include delayed presentation, non-specific clinical findings, lack of simple biochemical parameters that could be routinely used to diagnose the condition early, and time loss while performing tests for differential diagnosis in patients who are not immediately suspected to have AMI at presentation [3]. From a different perspective, if it is impossible to diagnose AMI in the early period in most patients, it becomes more important that parameters be determined that would be useful to predict the disease course at the time of diagnosis.